One of my favorite health tips drives my friends nuts when I start preaching about juicing!
Do you have enough time in your day to eat all the recommended fruits and vegetables that will keep you healthy and happy???
It's not easy! But my personal solution is MY JUICE MACHINE!
Look into getting a juicer for your own health boost! A juice machine is the best investment you can make for your health and happiness!
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What is melanoma skin cancer?
Melanoma skin cancer is the most serious form of skin cancer. If it is diagnosed and treated while it is still thin and limited to the outermost skin layer, it is nearly 100% curable. However once it advances and metastasizes to other parts of the body, it is difficult to treat and is often terminal. In the course of the past 10 years, the number of cases of melanoma has increased more rapidly than that of any other cancer. Around 51,000 new cases are reported to the American Cancer Society each year, and the probability of more unreported cases also exists.
What are its symptoms? Well malignant melanomas are generally small brown-black or larger multicolored patches, plaques or nodules with irregular outline. For early detection, one must keep in mind that a mole is suspicious if it is
* Getting bigger
* Changing shape, particularly getting an irregular edge
* Changing colour - getting darker,becoming patchy or multi-shaded
* Bleeding or becoming crusty
* Looks inflamed You should consult a doctor straight away.
1. Superficial spreading melanoma is the most common type, accounting for about 70 percent of all cases. As the name suggests, it travels along the top layer of the skin for a fairly long time before penetrating more deeply. The first sign is the appearance of a flat or slightly raised discolored patch that has irregular borders and is somewhat geometrical in form. The color varies, and you may see areas of tan, brown, black, red, blue, or white. Sometimes an older mole will change in these ways, or a new one will arise.
2. Lentigo maligna bears similarity to the superficial spreading type, as it also remains close to the skin surface for some time, and usually appears as a flat or mildly elevated mottled tan, brown, or dark brown discoloration. This type of in situ melanoma is found generally in the elderly, arising on chronically sun-exposed, damaged skin on the face, ears, arms, and upper trunk.
3. The third type, acral lentiginous melanoma, also spreads superficially before penetrating. However it is quite different from the others, as it usually appears as a black or brown discoloration under the nails or on the soles of the feet or palms of the hands. This type of melanoma is often found in dark-skinned people and hence common amongst African-Americans and Asians.
4. Unlike the other types, nodular melanoma is usually invasive at the time it is first diagnosed. The malignancy is recognized in the form of a bump. The color is often black, but occasionally is blue, gray, white, brown, tan, red, or skin tone.
Masni Rizal Mansor provide tips and review on melanoma skin cancer.
As always, before you attempt to self medicate or try a new health regimen or program we suggest you retain the services of a qualified health care professional.
Estrogen and Breast Cancer - The Evolving Mystery
As many women begin the transition through menopause a lot of consideration goes into the idea of taking Hormone Replacements - whether it is just Estrogen (Estrogen Therapy or ET) or a combination of Estrogen and Progesterone (Hormone Therapy or HT). For the purpose of this article Hormone Replacement Therapy (HRT) will refer to both HT and ET.
As well as more serious problems that may result from menopause like weakened bones, and osteoporosis.
Replacing your hormones with either natural or synthetic (laboratory made) forms of hormones may offer some benefit by helping to reduce many of the symptoms of early menopause - such as, hot flashes, insomnia, moodiness, and vaginal dryness. But as with EVERY pharmaceutical product made to help relieve pain or discomfort there are risks, and hormone therapy is not without them. What's important is that your unique health history and risk factors are discussed with your doctor to best determine whether or not HT or ET is the best option for you.
Estrogens Impact on the Breast
What you have to recognize is that the scientific community is just beginning to realize the uniqueness of every woman's hormone profile. The effect of HRT on one of your female friends may be vastly different from the effect they have on you. This is why there is such a large amount of variation in what each woman experiences as they transition through menopause. This is also one of the greatest challenges that Doctor's and Scientist face in their effort to understand the affects HRT has on the female breast.
Probably the biggest concern women have about HRT is the increased risk of that dreaded disease - Breast Cancer! Breast cancer risk, like many diseases, increases with age for women, but it is not specifically affected by menopause. Unfortunately, recent studies have generated a lot of conflicting data between HRT and breast cancer risk. But, before I go into the risk of developing breast cancers from HRT, it is important that you understand how Estrogen and Progesterone hormones affect your body, or more particularly, your breasts.
There are two primary thoughts on how cancer develops in the breast. One is that cancer tends to appear in fast growing tissue - Estrogen (and Progesterone) can cause breast tissue to grow at a faster rate - therefore this may be one way Estrogen is related to developing breast cancer. A second theory is that when estrogen is broken down in breast tissue the resultant compounds may bind to genetic material (DNA) and damage it. Damaged DNA is a common cause of cancers. For the moment - these are two primary theories on how HRT may increase the development of breast cancer.
Understand that the scientific community is hard at work trying to unravel the link between Estrogen and Breast Cancer -- or if there is even really a link -- but good science takes time. There are so many variables (like medical history and diet) in every study that it is impossible to blame ET or HT alone for breast cancer. Although the results of many clinical trials does show that HRT MAY increase the risk of developing breast cancer.
The Risk Factors
As a result of the various studies and clinical trials done, one thing is for sure, many more studies will need to be done before we unravel the mystery of HRT and breast cancer. In the mean time be aware of the fact that:
Breast cancer risk is increased with the use of ET, and to a greater extent with HT, use beyond 5 years. And observational data suggest a slight potential increase for breast cancer with HRT use for less than five years.
By how much the risk is increased is not exactly clear at this point and will likely vary depending on each woman's unique genetic makeup, diet and exposure to various environmental factors. Obviously, the risk factor of taking HRT has a much greater impact if you are already at risk of developing breast cancer. If you fit into any of the categories below - then most physicians would probably agree that you should avoid HRT.
One major consideration when contemplating HRT should be whether or not you are at a higher than average risk of developing breast cancer. Some other minor factors that may increase your cancer risk are weight gain and women who experience menopause later in life than average.
HRT - A Personal Decision
HRT is really a personal choice - there is no right or wrong answer. While some women may not want to accept the risks associated with HRT others may want to. Each woman must decide for herself, with input from her doctor, the best course of action. Both patient and doctor should be comfortable with the decision to take HRT or to pursue other treatments.
If you do decide to try HRT make sure you work closely with your doctor - prescribing the right dosage may sometimes turn into a bit of a guessing game. Keep your doctor informed as to how you are feeling and whether or not you think it is helping. The best thing you can do is actually keep a journal and track the symptoms that you're hoping HRT will help with. The lower the dosages and the shorter the duration is best if you are worried about increased breast cancer risk. But again your personal situation should be discussed in detail with your doctor, if you have another medical condition where the benefits HRT provide outweigh the increased breast cancer risk, then HRT should be considered.
If HRT is not for you - you are not without help. Thanks to all the ongoing research, new therapies and alternatives are being investigated every day and have proven successful for many women. But a word of warning - since this is a very new field, don't necessarily believe every thing you hear and read. There are a lot of companies trying to take advantage of this relatively new market. And since a lot of the herbal remedies are not regulated by the FDA some companies will package almost anything with no regard for scientific data to back up their claims. Look for resources that are impartial and companies that provide actual data to support what they are claiming.
Lastly, I personally am an advocate of a healthy diet and active lifestyle to beat menopausal symptoms and weight gain -- you should know that every clinical study I have read has shown that these two factors are proven to ease the menopause transition by regulating hormones naturally. And with no increased risk of breast cancer -- actually eating the right foods will help prevent breast cancer as well as many other types of cancer and disease. So regardless of your HRT decision -- the first step you should take when it comes to helping yourself is to eat a proper macronutrient profile and get that body of yours moving!
Jenny May and Health Thru Fitness, LLC focus on researching, developing, and implementing fitness and health strategies that the menopausal woman, as well as women of all ages, can apply to their current physical status. To learn more about the Jenny May Principles check out http://www.menopause-weight-gain.com and http://www.healththrufitness.com.
Lung cancer is a major killer of men and women across the country. Air pollutants, secondhand smoke, cigarette smoking especially, and other causative factors contribute to the disease. Most of us think that the lung cancer is mostly common among the cigarette smokers and it is true that the vast majority of cases are or were smokers. But there are non-smokers do get lung cancer and smokers who don't get it.
However it is better to identify lung cancer as possible. The most common early symptom of lung cancer is haemoptysis (coughing up blood). Consider this early symptom a warning sign of a cancer, which may be curable. If you find that you are coughing up blood, you should immediately contact your doctor , especially if you are 40 years of age, or older, with a long-term smoking habit.
Some of the primary signs include:
* Hacking, persistent cough
* Coughing up blood
* Shortness of breath
* Chest pain
* Lost appetite
* Lost weight
* Recurring bronchitis or pneumonia
The above symptoms also being the symptoms of many other lung problems, so it is always advisable to see a physician to ascertain the cause
Secondary signs of simple cell lung cancer include:
* Difficulty swallowing
* Changes in nails
* Hoarse or raspy voice
* High fever
* Swelling of facial features
It's true that most symptoms of lung cancer do not show themselves until the disease is in its advanced stages. However, sometimes people exhibit signs early in the disease's development. It is imperative that the first notice of symptoms or concern sends you straight to the doctor. The sooner treatment is begun, the better. A cure is possible if caught early enough and, if not, then a better quality of life and more of it.
When lung cancer spreads to other parts of the body, organs and bones, referred to as 'metastasizing', signs and symptoms include:
* Aching and sharp bone pain
* Changes in the brain that exhibit themselves through weakness, numbness, dizziness, and seizure
* Jaundice (whites of eyes, nails, and skin yellow)
* Masses near the skin's surface
* Numbness and loss of sensation in your extremities
All of these signs and symptoms of lung cancer may be caused by other health problems. The only way to know for sure is to consult a doctor as soon as possible.
Learn More About Smoking and Lung Cancer and Other Causes
The Essential Role Of Diet In Cancer Prevention
The body is made up of small units called cells. New cells are constantly produced to replace cells that have become worn out or damaged. New cells are also made during growth, e.g. during infancy and childhood. Normally, the body regulates the growth of new cells but occasionally abnormal cells are produced. These abnormal cells do not function properly and if they are not destroyed by the body's surveillance system, they may develop (mutate) and also rapidly increase in number, causing cancer.
The abnormal cells may also spread to other parts of the body and multiply there. Cancer can occur in different parts of the body. In the UK, the most common cancers in men are lung cancer, prostate cancer and colon cancer. The most common cancers in women are breast cancer, lung cancer and colon cancer. Diet has a greater influence on some types of cancer than others. The strongest links are with some cancers of the gastrointestinal tract e.g. of the mouth, throat, stomach and large bowel (colon). Dietary factors may protect against or reduce the risk of cancer.
For example, fruit and vegetables, consumed regularly, are thought to help reduce risk, whereas a low fibre intake or a high alcohol intake increase risk. Diet is only usually one of several factors involved. There is moderate evidence that higher vegetable consumption will reduce the risk of colon cancer and that higher fruit and vegetable intake will reduce the risk of stomach cancer. There is also moderate evidence that higher consumption of fruit can reduce lung cancer risk although the major risk factor is smoking. The more of these foods consumed, the greater the reduction in risk. At least five portions of a variety of fruit and vegetables per day is recommended. One of the mechanisms proposed to explain the effect of fruit and vegetables is via the antioxidants they contain, such as vitamin C, carotenoids and other plant phytochemicals.
However, although dietary intakes and blood levels of antioxidant nutrients such as vitamin E and beta-carotene have generally been associated with a lower risk of cancer (e.g. lung cancer), supplementation studies have not generally supported a protective effect.
In fact, supplementation with large doses of beta-carotene may increase the risk of lung cancer in high risk subjects (e.g. smokers). Maintaining body weight within the normal range may reduce the risk of some types of cancer. Being obese (very overweight) and having high intakes of energy may increase the risk of some cancers. For example in postmenopausal women, breast cancer risk is associated with being overweight.
Fat provides a large amount of energy so a high fat diet may make weight gain more likely. People who drink large amounts of alcohol have an increased risk of certain types of cancer, particularly liver cancer and cancers in the mouth and oesophagus. If such people also smoke, this makes the risk of cancer even greater. Carcinogens are substances which can start the process of cancer. Tobacco smoke contains carcinogens which cause lung cancer. Foods may also contain carcinogens. However, the risk from carcinogens in foods is low because if they are present at all, it is usually in very small amounts.
Carcinogens in foods may be substances that occur naturally, they may be due to contamination, or they be formed during cooking or processing (e.g. in smoked foods or foods that have been blackened during cooking e.g. barbecuing).
Aflatoxins are examples of carcinogens present in poorly stored foods. They are linked to mouldy produce, especially peanuts that are contaminated by the growth of moulds.
Research has shown a strong correlation between diet and cancer.
Get The Facts About Colon Cancer
The colon, or large intestine, is the last portion of the gastrointestinal tract and ends with the rectum. Cancer of the colon is the second most frequent cause of death from cancer in the United States. The risk of colon cancer increases as people age - 90 percent of colon cancers occur in individuals over 50 years of age. Colon cancer is also more common in people with a family history of colon cancer, or a personal history of colon polyps or inflammatory bowel disease. Cigarette smoking and excessive alcohol consumption may also increase the risk of colon cancer. Mutated versions of several genes have been linked to colon cancer.
For example, in their normal form the genes MSH2, MLH1, PMS1, and PMS2 correct tiny errors that occur when cells divide and grow.
Mutated versions of these genes cannot make such repairs, and eventually an accumulation of many such errors interferes with a cell's ability to resist the uncontrolled division and growth that characterize cancer. Research has linked the consumption of certain foods to colon cancer.The more red meat and animal fat that people eat, the greater their risk of developing colon cancer. Physicians continue to recommend low-fat, high-fiber diets as part of a healthy lifestyle to reduce the risk of high blood pressure, heart disease, and diabetes mellitus. Studies suggest that some drugs may decrease the risk of colon cancer.
For instance, estrogen replacement therapy after menopause and the use of nonsteroidal anti-inflammatory drugs such as ibuprofen appear to reduce the chances of developing this cancer. Colon cancer usually develops slowly and may not present apparent symptoms in its early stages. Some individuals with undiagnosed colorectal cancer may detect blood in their bowel movements (feces). They may also experience persistent constipation or diarrhea, abdominal pain, or unexplained weight loss. Two simple tests can detect most colon tumors while they are still in an early, easy-to-treat stage. The first test is the digital rectal examination, during which the physician uses a gloved finger to gently check the smoothness of the rectal lining.
The second test is the fecal occult blood test, in which a small sample of the patient's feces is smeared on a card coated with a chemical called guaiac, which reacts with blood. The card is analyzed in a laboratory for occult (hidden) blood. A positive result does not necessarily indicate the presence of cancer. The primary treatment for colon cancer is surgery to remove the tumor. The surgery may be combined with radiation, chemotherapy, or both. Using a combination of high-dose radiation and chemotherapy prior to surgery now makes it possible to avoid permanent colostomies in many patients who previously would have needed this procedure.
A colostomy is a surgical procedure to create an artificial opening through the abdominal wall to the exterior of the body for elimination of wastes into a plastic bag. If cancer has spread from the colorectal area to the lymph nodes or liver, surgery or chemotherapy used in combination with three drugs--fluorouracil, leucovorin, and irinotecan--prolongs the lives of some patients.
Cancer of the colon is the second most frequent cause of death from cancer in the United States.
This article was translated by mLingua Worldwide Translations, Ltd. - http://mlingua.pl
The demise of cells by programmed cell death referred to as apoptosis, a Greek word that means 'dropping off' or 'falling off' as in leaves from a tree, has been recently a topic of intense interest in biomedical sciences. Apoptosis is a well-defined sequence of morphological changes of cells that shrink and condense and then fragment, releasing small membrane-bound apoptotic bodies, which are phagocytosed by other cells. Importantly, the intracellular constituents are not released into the extracellular milieu where they might have deleterious effects on neighboring cells. On the contrary, cells that die in response to tissue damage or other reasons exhibit very different morphological changes generally called necrosis. The cells that undergo this process swell and burst, releasing their intracellular contents, which can damage surrounding cells and often cause inflammation.
Apoptosis refers to a particular morphology in which a chromatin condenses or coalesces to a heterochromatin in one or more masses in the nucleus. It usually settles along still-intact nuclear membrane referred to as margination of the chromatin. One of the essential functions of apoptosis is the elimination of cells in which DNA damages, faulty proliferation or improper adhesion to extracellular matrix that cannot be repaired. In cancer cells, the mechanism of apoptosis induction is broken. Therefore, more and more ideas and hypotheses for selective inducing apoptosis in cancer cells are tested in a growing number of laboratories all over the world.
The subject of programmed cell death has been recently discussed in almost 80 000 publications. As it is known, cell apoptosis may be induced by various stress factors (e.g. hypoxia, expression of oncogenes, mutations, DNA damages). On the other hand, apoptosis may be induced via internal or external signals, for instance proteins. Some of such endogenous and exogenous proapoptotic proteins have been found and described. Their genes may be used in modern anticancer therapies.
For example, introducing into cancer cells proapoptotic genes as Bax, Bcl-X5 or E2F-1 significantly increases induction of apoptosis. Some clinical trials concern therapeutic application of a 121-amino acids apoptin originated from chicken anemia virus (CAV). Recent data suggest that apoptosis induced by this protein involves caspases, a family of cysteinyl aspartate-specific proteinases. In vitro results show that apoptin is very active against cancer cells without inducing toxicity to normal cells. This tumor-specific effect may be explained by the nuclear localization of the protein in tumor cells required for its action. Moreover, apoptin is equally active, such as p53-mutant, Bcl-2-overexpressing or BCR-ABL-expressing tumor cells.
Other investigations showed that E4orf4 induces apoptosis in cancer cells by linking with 2A (PP2A) phosphatase. Unfortunately, induction of apoptosis by introducing genes encoding proapoptotic proteins has been little known. One possible mechanism is associated with destruction of mitochondrial membranes and, in consequence, disturbing electrons transport, oxidative phosphorylation and ATP synthesis. Finally, the cell dies but the death is slightly different than that during typical apoptosis induced by caspases due to prolonged time of this process. Proapoptotic proteins cannot be directly introduced to cancer cells because there are no specific receptors. They are transported through membranes in complexes by special fusion proteins called ligands.
Other method is introducing them as genes by vectors and this approach has been already successfully applied. Clinical trials are presently underway to test efficiency of new apoptosis-triggering drugs. A large number of adenoviral agents are being constructed, including replication-incompetent and replication-selective oncolytic adenoviruses. One of them is ONYX-015, a replication-competent virus genetically engineered to selectively replicate in and lyse p53-deficient cancer cells. Other agent, INGN 201, was shown to deliver a p53 expression. Preclinical studies in human cell lines and animals with head and neck cancers have shown that the p53 gene is transcribed and translated into p53 protein.
Respectively, 5% and 58% of patients receiving three intratumoral injections of INGN 201 in conjunction with radiation therapy for over 6 weeks were shown to have achieved complete and partial responses. Other example may be a gene encoding the proapoptotic Vpr protein that was successfully transferred into cancer cells by the HIV-1 virion. These agents are introduced by intravascular infusion or intratumoral or epitumoral injections. An example of a target therapy against cancer is an intravenous administration of liposomal form of tretinoin (ATRA). Treatment of acute promyelocytic leukemia (APL) with ATRA alone or in combination with chemotherapy results in an almost complete remission rate as high as 85% to 95%.
Other proapoptotic anticancer therapeutics is Genasense developed by the Genta Company. Genasense is a phosphothioate oligonucleotide consisting of 18 modified DNA bases. First, the single-stranded DNA molecule must be incorporated into a cancer cell and then target the mRNA by having a complementary sequence to it. This drug inhibits the production of a protein known as Bcl-2 that is widely expressed in many types of cancer. This up-regulation of Bcl-2 blocks the release of cytochrome C from the mitochondria thereby preventing apoptosis. Furthermore, Bcl-2 appears to be a major contributor to both inherent and acquired resistance to current anticancer treatments.
By inhibiting production of Bcl-2, Genasense enables the cancer cells to be killed by apoptosis when treated with current state of the art therapy. Interesting apoptosis-inducing drug is Velcade jointly developed by NCI and Millenium Pharmaceuticals. Activity of Velcade is mainly associated with reversible inhibition of the proteasome and building up many proteins including BAX. In the normal cells, the BAX protein induces apoptosis by blocking the activity of Bcl-2. When BAX level increases, BAX inhibition of Bcl-2 also increases and the cells undergo apoptosis. Non-clinical studies have demonstrated that cancer cells are more sensitive to the effects of the proteasome inhibition than normal cells.
Adachi, S.L.L., Carson, D.A., Nakahata, T., 2004.
Apoptosis induced by molecular targeting therapy in hematological malignancies. Acta Haematologica 111, 107 -123.
Ferreira, C.G., Epping, M., Kruyt. F.A.E., Giaccone, G., 2002. A
poptosis: Target of Cancer Therapy. Clinical Cancer Research 8, 2024-2034.
Ghobrial, I.M., Witzig, T.E., Adjei, A.A., 2005.
Targeting Apoptosis Pathways in Cancer Therapy. CA: A Cancer Journal for Clinicians 55, 178-194.
Hengartner, M.O., 2000.
The biochemistry of apoptosis. Nature 407, 770-776.
Lowe, S.W., Lin, A.W., 2000.
Apoptosis in cancer. Carcinogenesis 21, 485-495.
Tamm, I., Dorken, B., Hartmann G., 2001.
Antisense therapy in oncology: new hope for an old idea? Lancet 358, 489-197.
Tamm, I., Schriever, F., Dorken, B., 2001.
Apoptosis: implications of basic research for clinical oncology. Lancet Oncology 2, 33-42.
Radoslaw Pilarski is a PhD candidate working on anticancer properties of Uncaria tomentosa - http://www.uncariatomentosa.com - at Institute of Bioorganic Chemistry (Polish Academy of Sciences, Poland). mLingua Worldwide Translations, Ltd. - http://mlingua.pl - provides professional language translations to and from all major Western and Asian languages, software localization and web site translation services.
Today's woman is more knowledgeable and conscientious than ever about the risks of breast cancer. This is especially true for women over 40 years of age, who have crossed the threshold of increased risk of breast cancer. Age is so important to the development of breast cancer that about 76% of women who develop it have no other risk factors other than age. However, all women, regardless of age or race, need to acknowledge the risk of developing it. All women are at risk. Women who have never smoked a day in their entire lives can develop breast cancer. Women who have been always been health conscious can also develop it. In fact, nearly one woman out of eleven will experience breast cancer. A staggering statistic by anybody's standards!
Three of these begin in situ and sometimes become invasive; the fourth, most dangerous is invasive from the start. It is handy to recognize the names and characteristics of each type.
An estimated 211,000 new cases of breast cancer was diagnosed this past year. Even with the increased awareness programs, early detection through annual mammography screening and instruction for self-examination, breast cancer remains a leading cause of death for women. Breast cancer, like other forms of cancer, is a disease of the cells. In all, there are about fifteen different types of breast cancer. Some are more serious than others, but the one common factor each shares is that neither the cause nor the cure has been found.
There are four recognized developmental stages of breast cancer:
(1.) State 0: Cancer cells are present in either the lining of the milk glands (lobules), or in the tubes (ducts) that link the milk glands to the nipple. No cancer cells have spread to the nearby fatty tissue.
(2.) Stage 1: Cancer has spread to nearby fatty tissue in the breast. Tumor size is about 1" or under; no cancer cells are present in surrounding lymph nodes.
(3.) Stage 2: Size of tumor is 1" to 2" in diameter; cancer cells may have also spread to nearby lymph nodes.
(4.) Stage 3: Cancer is locally advanced. Tumors are approximately 2" or larger in diameter, or tumors of any size have spread to lymph nodes under the arm or in the chest (above or below the collarbone).
(5.) Stage 4: Metastatic, advanced breast cancer. The cancer has spread from the breast and lymph nodes to other parts of the body.
Early detection of breast cancer remains a woman's best chance of survival, and women of all ages should take advantage of all the resources available.
Every woman should:
1.) Become educated about the risk factors associated with breast cancer.
2.) Become knowledgeable about the types, stages, and symptoms of breast cancer.
3.) Learn the correct procedure for self-examination tests, and perform them routinely.
Long-term use of oral contraceptives, early menstruation, late first full-term pregnancy, exposure to high doses of radiation - puberty through childbearing years, and inherited genetic mutation can all increase a woman's risk of acquiring breast cancer.
Women 40 years of age and older should also:
1.) Have an annual mammography screening.
2.) Become educated about increased age-related risks associated with breast cancer.
Recent studies confirm that risk of breast cancer in midlife increases with regular consumption of alcohol, hormone replacement therapy, weight, and body mass distribution. During self-examination, look for a lump or thickening in the breast, a discharge from the nipple, scaliness on the skin or around the nipple, a change in shape, color, or texture, and dimpling or puckering. If you detect a lump, don't panic. About 85-percent of all lumps turn out to be nonmalignant. Make an appointment with your doctor for a more thorough examination, and tests. Treatment for breast cancer today is often less radical than in years past, and chances for survival much better when the tumor is discovered early.
Author's note: The third Friday in October is National Mammography Day. This year, that day will be celebrated October 20, 2006.
Lori S. Anton is a professional writer and is founder of Writers Write Now, offering original SEO content, quick content, and free content for web sites. She lives in rural Wyoming with her husband, Jeff, and their diabetic canine companion, Muffy. Lori is also pet editor for the Savvy Dog Lover web site - where the be best bones are buried!
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